Regarding suppression of the tumor function by dasatinib, in function studies of ASPS, a genome-wide location analysis of ASPS tumor samples and cell lines expressing ASPSCR1–TFE3 defined a subset of approximate 400 genes as putative regulated direct targets of ASPSCR1–TFE3, including c-MET [17]. This evidence concerns the gene ASPSCR1 and neoplasm.