Post-transplanted proteinuria could be originated from several causes including factors related to graft lesions during transplant procedure, such as long cold ischemia time; factors related to the recovery of graft function, such as delayed graft function, acute rejection episodes, and effect of mammalian target of rapamycin inhibitor drugs; factors related to functional discrepancy between the donors and recipients; and factors related to donor characteristics, such as age and cardiovascular diseases [12, 14, 18, 19]. The gene discussed is MTOR; the disease is cardiovascular disorder.