Since IFNγ primes phagocytes such as monocytes for increased oxidative burst while MIG/IP-10/RANTES act as chemoattractants for monocytes/macrophages, natural killer cells or T cells, we investigated whether type 2 DM PBMCs with poor glycemic control have defective capacity to control hvKP infection relative to healthy PBMCs. The gene discussed is IFNG; the disease is infection.