From these data, the authors concluded that ARH‐I can drive cancer cell dormancy in the presence of factors that promote survival in the cancer microenvironment through modulation of autophagy.22 From a clinical point of view, these findings suggest that relapse of ovarian cancer may result from the breakdown of dormancy induced by changes in the extent of CAFs infiltration in the tumor stroma. This evidence concerns the gene TBX1 and ovarian cancer.