CYGB and Hepatic fibrosis: Finally, Pan et al. (41) showed an opposing function between low- and high-molecular-weight FGF2,and the administration of low-molecular-weight FGF2 effectively reversed liver fibrosis.In addition to the previous study results obtained using rodent models and cells, werevealed here that FGF2 is a key molecule for CYGB induction and for maintaining humanHSCs in a quiescent-like phenotype.