As the two tables showed, there were more hypertension, a higher proportion of urine sediment, higher levels of SUA, blood urea nitrogen (BUN), serum creatinine (Scr), triglycerides, blood glucose, phosphorus, parathyroid hormone (PTH), 24 h urinary albumin, 24 h urinary α1-microglobulin, urine N-acetyl-β-D-glucosaminidase (NAG) activity, lower levels of eGFR, serum album, complement 3 (C3), 24 h urinary calcium, urinary volume and urinary pH in LN patients with HUA than those without HUA. This evidence concerns the gene ALB and hypertensive disorder.