Indeed, except for p-T421/S424-p70S6K, p-T308-Akt/PKB, p-Y216–GSK3β and p-T202/Y204-ERK1/2 were significantly higher in DM1 TA than in CTR TA (p-T308-Akt/PKB, CTR vs DM1 p = 0.04; p-Y216–GSK3β, CTR vs DM1 p = 0.004; p-T202/Y204-ERK1/2, p = 0.03). This evidence concerns the gene GSK3B and myotonic dystrophy type 1.