Based on our previous reports on the immunopathological roles of IL-17A and our current results that IL-17A originates from γδT cells during the early stage of infection, the immunopathological roles of γδT cells, as well as the Vγ1+γδT and Vγ4+γδT subsets, were further explored in the early phase of severe influenza virus infection. This evidence concerns the gene IL17A and infection.