EGFR and erythroleukemia: It is likely that another oncogene such as v-erbB (a mutated version of epidermal growth factor receptor; EGFR) would be needed, as in AEV-induced erythroleukemia and sarcoma, to collaborate with TRα1PV to bring out the transformed phenotypes in erythroid cells of Thra1PV/+ mice.