We recently reported FGF-2 regulates the progression of corneal neovascularization beyond resolution of infection (14–21 days PI, latent infection) and its neutralization suppresses the expression of proangiogenic factors and preserves visual acuity without altering corneal sensation.45 Of note, in the present study we found FGF-2 was highly expressed in the UI cornea and its content not modified as a result of acute infection (6 days PI) or CSF-1R+ cell depletion. The gene discussed is FGF2; the disease is infection.