For example, human monoclonal thyroid-stimulating hormone receptor (TSH-r) has been shown to cross-react with Yersinia enterocolitica (Y. enterocolitica), thereby providing a mechanistic framework for molecular mimicry in Graves' disease, where Y. enterocolitica antibody production promotes cross-reactive pathogenic response to TSH receptor [6]. The gene discussed is TSHR; the disease is Graves disease.