By comparing data of PAS-3 patients with only Hashimoto’s thyroiditis (whose THAb positivity rate is three out of 10), we infer, as mentioned earlier, that the presence of NTAID in the background of Hashimoto’s thyroiditis adds little influence on the propensity to form THAb, with negligible influence of associated NTAID on thyroid hormones bound by THAb and classes of immunogloblulin. Here, TG is linked to Hashimoto thyroiditis.