Specific examples include the pro-inflammatory cytokines IL-6 and MCP-1 which associate with worse clinical outcomes [3] Conversely, the anti-inflammatory cytokine, IL-10, has been linked with susceptibility to respiratory infections and sepsis-associated mortality [4, 5] Hence, there is a medical need for new therapeutics that modulate host responses following burn injury [6]. Here, CCL2 is linked to Sepsis.