In attempt to dissect whether CCL3 influences the tumour milieu, we evaluated the expression of molecules involved in tumour proliferation (EGF, FGF, TGF-β and TNF-α) [36, 37] and angiogenesis (TGF-β1, VEGFa and VEGFb) [38–40]. The gene discussed is VEGFB; the disease is neoplasm.