However, there was no significant difference in the number of NeuN+ neurons between iNSC treated and normal control animals (169 ± 115 cells/mm2 vs. 278 ± 9 cells/mm2, respectively) at the lesion border, indicating that iNSC treatment leads to neuronal protection at the lesion site post-stroke (Fig. 6A–D). The gene discussed is RBFOX3; the disease is stroke disorder.