RBFOX3 and stroke disorder: These studies showed that hESC- and fetal-derived NSC treatment enhances cell proliferation and migration in the SVZ, DG, and striatum, and a subset of these cells co-label with the neuroblast marker DCX and the mature neuron marker Fox3, indicating endogenous neurogenesis mediated by NSC transplantation may contribute to neuronal replacement in the stroke-damaged brain33–39.