CD8A and graft versus host disease: Broadly expressed minor histocompatibility antigens (mHag) e.g. H-Y have been identified as the in situ targets of CD8+ T cells mediating GvHD.[36] However, only few GvHD associated mHags have been molecularly characterized to date.[37, 38] Therefore, we performed our analysis of miR-625-3p in the overall CD8+ T cell population assuming that these may comprise sufficient mHag specific T cells to allow detection of GvHD related miR-625-3p regulation.