Expression of wild-type Nramp1 thus promotes an inflammatory cytokine response following infection with B. thailandensis (as well as in response to S. Typhimurium infection and LPS stimulation as controls) which was not observed following infection with B. pseudomallei (Figure 3) suggesting that this pathogen has a strategy to avoid the influence of functional Nramp1. The gene discussed is SLC11A1; the disease is infection.