LTBR and parasitic infectious disease: The failure to mount an effective specific T and B cell response against T. gondii and the possible inability to drive the parasite into its chronic stage and/or to prevent reactivation of chronic toxoplasmosis might explain the higher parasite numbers observed in the brains of LTβR−/− animals and concurs with the increased parasitemia described in LTβR−/− animals in the ECM model by other groups [53, 54].