SEMA3A and retinopathy of prematurity: In a similar manner to Sema3A, Wei et al. [123] recently showed that absence of neuronal Nrf2, a major stress-response transcription factor responsible for cell-intrinsic cytoprotective function, results in Sema6A induction in hypoxic/ischemic retinal ganglion cells that diminished normal revascularization into the avascular zones in the inner retina from ROP animals.