Similarly, mean prolidase activity was lower among diabetic acute MI subjects compared with both diabetic and non-diabetic stable CAD subjects at both the acute and quiescent time points [p < 0.05 as evident from non-overlapping 95% CI of the mean (Table 5) and diabetes effect p < 0.0001; MI × diabetes p = 0.02 (Figure 3)]. This evidence concerns the gene PEPD and coronary artery disorder.