Previous studies have reported that inhibition of DPP‐4 activity in the blood leads to an increase in the circulating number of EPCs 7, 8, 9, enhancing angiogenesis and blood flow in hind limb ischaemia (HLI) 8, 9 and protecting the heart in models of myocardial infarction 10, 11. The gene discussed is DPP4; the disease is myocardial infarction.