Moreover, analysis of cytotoxic molecules involved in CTL-mediated antitumor immune response (FasL, perforin, and CD107) showed that MSCs managed to attenuate influx of FasL+ CTLs (Figure 4(b), p < 0.05), perforin + CTLs (Figure 4(c), p < 0.05), and CD107+ CTLs (Figure 4(d), p < 0.01) in the lungs of tumor-bearing animals. The gene discussed is PRF1; the disease is neoplasm.