In this way, we determined multiple connections of the FOXP2-driven network and its LCC to developmental (ASD, SCZD, Down syndrome, agenesis of corpus callosum, trismus-pseudocamptodactyly, ankyloglossia, facial dysmorphology) and neurodegenerative disorders and diseases (AD, PD, HD, LBD, ALS), deafness, and dyslexia (for details, see Discussion). This evidence concerns the gene FOXP2 and Huntington disease.