Chlamydial infection is known to activate innate immunity signaling pathways, including those mediated by MyD88 [14, 26] and STING [27, 28] and to induce inflammatory cytokines [12, 29] and both cellular [30–33] and soluble effectors [34, 35]. The gene discussed is STING1; the disease is chlamydia trachomatis infectious disease.