This means that there is possibility that the interaction of hsa-miR-96 and the oncogenic mRNAs mediated by other genes, or the up-regulated oncogenic mRNAs affected hsa-miR-96 to be over-expressed, and the overexpressed hsa-miR-96 suppress FOXO1 and FOXO3a to promote tumor proliferation and invasion. The gene discussed is FOXO3; the disease is neoplasm.