The persistence of this NKG2C+ subset several months after a CMV reactivation was one reason to propose them as an adaptive or memory-like form for NK cells.8 In the CMV model, the memory-like CD57+ compartment is replenished through the maturation of effector NKG2C+ NK cells, several months after the acute phase of infection, once viremia is controlled.8 The CMV-DNA levels were however not above the reactivation threshold in the patients of this study, suggesting that CMV reactivation did not occur here during PHI. This evidence concerns the gene B3GAT1 and infection.