Thus, it is possible to propose that the development of autoimmune processes in the EAE mice after their immunization with DNA (more similar to those in the case of SLE) and delay in the development of autoimmune processes comparing to that in the case of mice treatment with MOG may reflect the non‐universality of the genetic predisposition of these mice only to MS. Here, MOG is linked to systemic lupus erythematosus.