Because recent reports have demonstrated that ZIKV can infect primary neuronal cells, and ZIKV infection of neurospheres and the CNS is a major hallmark of brain deformities, abnormalities, microcephaly, or other detrimental effects [2, 3, 6, 32, 33], we sought to determine if Jak-STAT pathway inhibition may be modulating infection in human neuroblastoma cells. Here, SOAT1 is linked to infection.