Such an initial type I IFN response might induce chemokines, including IFN-γ-inducible protein 10 (IP-10)/CXCL10, which in turn could recruit cells expressing the corresponding chemokine receptor CXCR3, such as T, natural killer (NK), and NKT cells, to the site of infection, which might contribute to the local immune response by IFN-γ secretion (Figure 2; Liehl et al., 2014; Miller et al., 2014). Here, IFNG is linked to infection.