Consistent with previous studies, our results showed that expression of these inflammatory cytokines significantly increased in the skin lesions and sera of IMQ-treated mice compared with those of the normal group, and similar observations were made regarding the expression of IL-17 and IL-23, which are critical for the development of IMQ-induced psoriasiform dermatitis, whereas the administration of BML-111 significantly downregulated the expression of most of these cytokines. Here, IL17A is linked to Psoriasiform dermatitis.