The absence of dystrophin in the muscle cells results in progressive muscle weakness and muscle damage which is reflected by changes in energy metabolism, inflammation, fibrosis and eventually by progressive replacement of muscle tissue by fat.[1] Although numerous clinical trials have so far led to conditional approval of a few compounds by the Food and Drug Administration (FDA) and European Medical Association (EMA), there is still an urgent need for more effective therapies and outcome measures in DMD.[2]. This evidence concerns the gene DMD and Duchenne muscular dystrophy.