Furthermore, we have determined that E-cadherin, a direct and functional target of miR-151a, can potently inhibit NSCLC cell migration and the transition to a mesenchymal-like cell phenotype, indicating that miR-151a-induced E-cadherin repression is a primary mechanism by which miR-151a enhances partial EMT of NSCLC. Here, CDH1 is linked to non-small cell lung carcinoma.