Finally, even though CRP, hsCRP, and IL-6 were not associated with self-reported diabetes in our study, higher levels of receptors for pro-inflammatory cytokines, including IL-6 (sIL-6R) and tumor necrosis factor-alpha (TNF-α) (sTNFR1 and sTNFR2), showed trends for associations with self-reported diabetes, findings which are supported by prior literature for sIL-6R [19, 25] and either sTNFR1 or sTNFR2 [23, 26, 29, 35]. The gene discussed is TNF; the disease is diabetes mellitus.