IFNB1 and myasthenia gravis: IFN-I, especially IFN-β, could play a central role in the thymic follicular hyperplasia of MG patients by inducing high expression of α-AChR and of CXCL13 chemokine in thymic epithelial cells, and of the chemokine CCL21 in endothelial lymphatic cells, two chemokines involved in the abnormal recruitment of B cells in EOMG thymuses.