ASCT2 expression was high in 43.6% (17 of 39) of CRC patients with mutated KRAS, whereas in 24.1% (13 of 54) of CRC patients with wild-type KRAS, which indicated that there was a significant correlation between high ASCT2 expression and KRAS mutation (risk ratio: 1.62, 95%; confidence interval (CI): 1.02–2.57, p = 0.047, Figure 2b). This evidence concerns the gene SLC1A5 and colorectal carcinoma.