One patient with influenza‐associated transverse myelitis was found to have high titers of myelin oligodendrocyte glycoprotein antibodies, suggesting a pathogenic role.137 Additionally, one patient had recurrent ADEM after a second influenza infection 6 months after his first episode and another had recurrent ADEM after an influenza vaccination suggesting a role for genetic predisposition or formation of an immunologic mechanism.132, 135. The gene discussed is MOG; the disease is acute disseminated encephalomyelitis.