One study of autopsy findings in five A(H1N1)pdm09 cases found evidence of ATN in all patients.190 Similarly, in a study of 21 patients who died with A(H1N1)pdm09 infection in Brazil, all patients demonstrated mild‐to‐moderate ATN; four of those patients also had myoglobin pigment in the tubules consistent with rhabdomyolysis, and a fifth had evidence of thrombotic angiopathy.191 Direct viral injury to the kidneys is another postulated mechanism of disease, but there is limited evidence to support this. The gene discussed is MB; the disease is oculocutaneous albinism type 1.