While the DCDC2 deletion is interesting for the potential regulatory effect, other DCDC2 markers, mainly SNPs, showed more consistent associations across independent studies.4, 5, 6, 7, 8, 9, 10 Our current study, therefore, does not undermine the overall evidence in support of DCDC2 as a candidate gene for dyslexia or reading abilities. The gene discussed is DCDC2; the disease is dyslexia.