It has been proposed that quercetin can influence the activation of epidermal growth factor receptor (EGFR) through the overexpression of some ligands like amphiregulin [12] and also has been shown that quercetin, at a dose of 10 μM, decreases the phosphorylation of EGFR in prostate cancer cells (PC3), producing a diminished expression of target proteins like vimentin, N-cadherin, and cyclin D1 [13]. The gene discussed is EGFR; the disease is prostate carcinoma.