CaMKII (calcium/calmodulin-dependent protein kinase type 2) is proposed to regulate inflammation (Cfb, Tnfa, and Il-6) and cardiomyogenesis in response to hypertension-related pressure overload, β-adrenergic agonists, or myocardial infarction-induced cell injury.30 Thus, Cfb may contribute to both cardiac inflammation and hypertrophy in response to stress, possibly through regulation of cardiomyogenic gene expression. Here, CAMK2G is linked to myocardial infarction.