This result is consistent with the findings of another study of B-cell lymphoma that showed that LMP1 promoted GLUT1 plasma membrane localization via IKKβ expression and activation of the NF-κΒ signaling pathway, which promoted EBV-infected B-cell lymphoma proliferation and apoptosis resistance by driving glucose import and increasing glycolysis [46]. This evidence concerns the gene PDLIM7 and B-cell non-Hodgkin lymphoma.