Recently, Ma et al. reported that CIK cells armed with BsAb CD3 × EGFR (EGFRBi-Ab) to target EGFR-positive glioblastoma significantly increased CIK cells cytotoxic activity in vitro and inhibited the growth of glioblastoma tumors in glioblastoma xenograft mice (87). The gene discussed is EGFR; the disease is glioblastoma.