In order to examine if HuR/TTP axis contributes to the pathophysiology of endometriosis, we evaluated expression of Zfp36, Zfp36l1, Zfp36l2 and Elavl1 in eutopic and ectopic lesions obtained from syngeneic BALB/cByJ mouse model of endometriosis. The gene discussed is ELAVL1; the disease is endometriosis.