Likewise, kininogen 1, alpha-2-macroglobulin, apolipoprotein A-I, and alpha-1-antiproteinase E have also been sporadically reported to be increased in infectious diseases [55], but their concomitant association which appeared specific during neurological clinical signalmen of E. cuniculi active infection is novel in such a context. This evidence concerns the gene KNG1 and infectious disease.