So far EMILINs have been mostly studied in the context of cardiovascular diseases since genetic ablation of EMILIN-1 in mice results in structural alterations of elastic lamellae in the aortic wall and hypertension5, 15, while EMILIN-2 null mice show suppressed platelet aggregation and delayed clot retraction18. The gene discussed is EMILIN2; the disease is cardiovascular disorder.