In both myeloid and vascular cells, FHL2 also plays an important role in atherosclerosis by promoting proinflammatory chemokine production, adhesion-molecule expression, and proinflammatory monocyte recruitment [13], and FHL2 deletion attenuates the formation of atherosclerotic lesions induced by a cholesterol-enriched diet [14]. Here, FHL2 is linked to atherosclerosis.