IL17A and infection: In some infection models, including C. muridarum, IL-17complemented the protective role imparted by the IL-12/IFN-γ axis through theinvolvement of myeloid differentiation factor 88 (MyD88) signaling whereMyD88-deficient infected mice showed reduced IL-17 responses along with reducedneutrophil infiltration, which is important for early control of diseasepathogenesis 87, 88.