Three additional predicted cancer driver mutations (EPHA7, MAP3K12, and PCSK5) were identified that were acquired during the transformation of non‐malignant MCF10A cells to malignant DCIS.com cells that could also be implicated in the cell‐context dependency of SOX11 in promoting invasion 31. The gene discussed is PCSK5; the disease is ductal breast carcinoma in situ.