In contrast, in patients with known NAFLD and compared to abstainers, a low to moderate consumption of alcohol has been associated with lower values of ALT [21], lower prevalence of hepatic steatosis [22, 23•, 24, 25], non-alcoholic steatohepatitis (NASH) [26], carotid plaques [27], and lower stages of fibrosis [26, 28•]. This evidence concerns the gene GPT and metabolic dysfunction-associated steatohepatitis.