In a similar biological process to that which is characteristic to PTSD (Williamson et al., 2015), ongoing secondary traumatization symptoms throughout childhood and adulthood may relate to unmodulated levels of glucocorticoids and catecholamines in the hypothalamic-pituitary-adrenal-axis (HPA axis) and the autonomic nervous system, resulting in the dysregulation of blood pressure, heart rate variability, and insulin, glucose and lipids metabolism. The gene discussed is INS; the disease is post-traumatic stress disorder.