To further investigate the clinical significance of miR-210-3p-induced TNIP1 and SOCS1 downregulation and the subsequent activation of NF-κB signaling in PCa tissues, miR-210-3p expression and the protein expression levels of TNIP1, SOCS1 and nuclear p65 were examined. The gene discussed is SOCS1; the disease is posterior cortical atrophy.